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High-Fat Diet Protects The Brains Of Fruit Fly From Concussions

According to a new study, a high-fat diet decreases male fruit fly aggression following concussions. The findings hint at promising treatments to avoid brain damage following head injuries. Researchers from the UConn (University of Connecticut) reported these findings in the journal Frontiers in Neuroscience. Concussions became a topic of discussion when NFL (National Football League) players successfully charged the league over alleges that recurrent head impacts due to playing professional football had eternally impaired their brains. It soon became apparent that players in other sports—professional and amateur—were also in jeopardy. And despite everything the dangers of concussion are now known, there is no best treatment for it other than to rest.

High-Fat Diet Protects The Brains Of Fruit Fly From Concussions

Derek Lee (CLAS)—Graduate Student from the UConn—said, “The treatments for concussion are quite non-existent.” One of the major and complicated aspects of examining concussions is that it is not completely clear how concussions impair the brain, nor why some damages are permanent and some not. Researchers found clues that concussions may kill brain cells by excitotoxicity. Excitotoxicity refers to the cells that get over-excited due to some incident or injury, and utilize all their glucose, the sugar they utilize for energy, too fast that sometimes the cells die. But glucose is not the brain cells’ only source of energy. These cells can also function on ketone bodies formed from fats.

Similarly, UConn was in news as its researchers found that the delivery of healthy donor cells is a solution for the correction of bone disorder. OI (osteogenesis imperfecta) is a set of genetic disorders that largely affect the bone. The individuals having OI have bones that rupture effortlessly, sometimes without an obvious cause. This disorder is mostly caused by mutations linked to type I collagen or molecules that contribute to collagen development, which causes defective collagen bone matrix. The existing treatments for OI target to correct the faulty bone matrix, but fail to aim at the basic collagen defect. The research was published in the journal Stem Cells.

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